Acidotic coma

Hyperosmolar non-keto-acidotic diabetic coma. A report of three cases and review of the literature.

Three further cases of hyperosmotic nonketo-acidotic and non-lactic-acidotic diabetic coma have been described. A favourable outcome in these patients emphasizes the importance of recognizing this condition in its early stages. This may be difficult as there is no ketosis and no hyperventilation-features that one normally associates with diabetic coma. The signs are those of increasing drowsine...

Subacute necrotizing encephalopathy in an acidotic child.

Subacute necrotizing encephalopathy was described originally by Leigh (1951). Further case reports reviewed by Ebels, Blokzijl, and Troelstra (1965) have confirmed this condition as a neuropathological entity but ideas concerning its aetiology have remained speculative. Recently Worsley, Brookfield, Elwood, Noble, and Taylor (1965) have reported a case, with confirmation at necropsy, in which t...

Myxedema coma

RAIONALE Myxedema coma (MC) is extremely rare but lethal in pediatric patients with hypothyroidism leading to altered mental status and hypothermia. But there is no clinical guideline for such cases. PATIENT CONCERNS A 6-year-old Chinese girl presented with coma and hypothermia preceded by pneumonia. Her lab results were: free thyroxin (T4) 4.18 pmol/L and thyroid-stimulating hormone (TSH) > ...

Coma Cluster

Coma Blisters

Coma blisters are lesions that occur in the setting of a variety of neurological diseases. Although most commonly associated with barbiturate overdose, they can be seen in the setting of coma due to other etiologies. Blisters develop 48-72 h after the onset of unconsciousness. We report the case of a 29-year-old man who presented to the emergency department with confusion and progressive loss o...